They affect mostly both eyes, but the types of defect can combine and their intensity can differ. Defects of choroid and colobomas are distinguishable in 8 - 12 weeks. In this case we talk about "severe form of CEA", which is present in about 25 % of animals suffering from CEA. When only iris is affected, there are no blind spots, but retinal detachment can cause the blindness of the eye. Colobomas (clefts) of the optic nerve and the eyeball can also be present, resulting in missing parts of iris or retina. In case of ectodermal involvement, tortuosity of blood vessels, retinal folds and detachment, and/or intraocular bleeding can be observed. Animals affected by mid-severe CEA won´t go blind in most cases, but their offspring can develop severe forms of the disease. That is why it is necessary to perform clinical examination of puppies in at early age. After three months of age, retinal pigment can cover the defective choroid. The number of blood vessels is reduced and they can have abnormal shapes. When examined by an ophthalmoscope, little or no retinal pigment displays underlying blood vessels and sclera. In this case we talk about "mid-severe CEA". In case of mesodermal defect choroid hypoplasia develops, which is considered the primary characteristic of CEA. Postnatally, different defects are found depending on the severity of disturbances in three layers of the eye. The most often affected breeds include Rough Collie, Smooth Collie, Border Collie, Shetland Sheepdog, Nova Scotia Duck Tolling Retriever and Australian Sheepdog.ĬEA is caused by an abnormal development of the eyeball, starting at about day 30 of the embryonic development. * The tests are performed by the partner laboratory. Genomia guarantees the quality of its partner's services.Ĭollie Eye Anomaly (CEA) is an inherited eye defect.
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